Glycogen synthase kinase (GSK) 3beta hyperactivity impairs glomerular podocyte insulin signaling via IRS1 modulation in diabetic kidney disease

Authors

  • Mengxuan Chen The University of Toledo Department of Medicine Author
    • Yan Ge Division of Nephrology, University of Toledo College of Medicine Author
      • Lance Dworkin Division of Nephrology, University of Toledo College of Medicine Author https://orcid.org/0000-0002-2471-5989
        • Rujun Gong Division of Nephrology, University of Toledo College of Medicine Author

          DOI:

          https://doi.org/10.46570/utjms-2025-1352

          Keywords:

          GSK3beta hyperactivity, hyperinsulinemia, diabetic kidney disease
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          Published

          2025-06-30

          Issue

          Section

          Dr. Lance D. Dworkin Department of Medicine Research Symposium

          How to Cite

          1.
          Chen M, Ge Y, Dworkin L, Gong R. Glycogen synthase kinase (GSK) 3beta hyperactivity impairs glomerular podocyte insulin signaling via IRS1 modulation in diabetic kidney disease. Translation. 2025;13(S3). doi:10.46570/utjms-2025-1352

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